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Buspirone purchase has also been shown to increase brain serotonin buy generic buspirone availability. The pharmacologic effects of Order clomid cheap antidepressant drugs result from the modulation of serotonin concentrations in the CNS [13]. pharmacologic interaction between tianeptine and antidepressant medications is buspirone hcl generic mediated by the serotonin transporter. SSRI antidepressants fluoxetine and venlafaxine have been shown to increase the concentration of transporter in brain [14]. Several studies have shown that acute tianeptine dosing increases the systemic concentration of serotonin transporter in humans [8,10,13]. It is thought that the initial increase in amount of the serotonin transporter reflects direct mechanism of action tianeptine (and its metabolite 9-phenyl-1-piperdine) and other SSRI antidepressants. These studies were largely controlled in their design. It is known that the first 50–200 mg of tianeptine or its metabolite 9-phenyl-1-piperdine administered daily induces a small but sustained rise in the brain serotonin concentration. Most of the increased activity is sustained until the next tianeptine dose is taken; however, there are several exceptions [15]. These include case reports [16,17] and single observations [18,19]. Thus, there is reason to believe that short-term tianeptine administration is not detrimental for brain serotonin levels. In the long term, increased concentration of the serotonin transporter leads to a net decrease in the systemic serotonin concentration. If transporter is reduced or eliminated, therefore, there is an increase in the concentration of monoamine oxidase inhibitors. This effect of these medications on serotonin levels in the CNS has been described in several clinical studies [9]. Fluoxetine increases the cerebral serotonin concentration by 5–15% on a low to medium dosage [10]. Vincristine blocks serotonin-induced release of 4-hydroxy-5-methoxytryptamine, a major metabolite serotonin metabolites, but has not been reported to block serotonin-induced release of dopamine. Because both Vincristine and Fluoxetine are in the drugs' mechanisms of action and have been reported to affect serotonin levels in some patients, we did not include them in the analysis of changes serotonin levels in this study. MATERIALS AND METHODS Subjects were recruited by advertisement and written consent. A random sample of 50 healthy volunteers participated in these studies. All subjects had regular sleep, medication, and dietary, exercise, medical histories. Subjects were not encouraged to undertake any special programs in order to reduce intake of noradrenaline (N.E.A., 1988). After having completed all screening and baseline assessment, subjects were randomly assigned to receive tianeptine by mouth (TID) or placebo (P). All subjects were instructed to take the medication on an empty stomach as instructed by the investigator, and no one was allowed to eat. The TID dose of tianeptine was 50–100 mg, with a half-life of 2.5–6 hours in healthy volunteers (D.H.B., unpublished observations). The dosage was selected based on several criteria. It consisted of a lower dosage than the usual daily dose of average, well-cons.

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